Letter to the Editor
Metformin-associated Lactic Acidosis in a Patient with Vertebral Artery Dissection
Abstract
Metformin, a biguanide antidiabetic drug, has been implicated as a cause of lactic acidosis, usually in a setting of renal failure, IV contrast administration, shock, sepsis, hypoxemia, heart failure, or liver disease. Although metformin can cause mild lactic acidemia in type 2 diabetes mellitus patients with normal renal function,1,2 there are very few reports of lactic acidosis (blood lactate >5 mEq/L, anion gap >15, arterial pH <7.35) in patients with normal renal function and no other obvious precipitating cause. In the following case, a metformin-treated man with mild and limited stroke symptoms and normal renal function developed lactic acidosis in the absence of other risk factors.This content is limited to qualifying members.
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