Abstract

AbstractAcute pancreatic inflammation results in pain, tenderness, nausea, vomiting, and exudation of intravascular fluid into extravascular compartments. An edematous reaction or a more destructive process may occur. Chronic alcoholism and cholelithiasis are the most common precipitating factors. Most cases are easily diagnosable if pancreatitis is considered. However, pancreatitis may mimic other abdominal emergencies. Important aids in diagnosis include a compatible history, markedly elevated serum and urine amylase values, elevated serous cavity fluid amylase, and the presence of methemalbumin. The primary goal of treatment is reduction of pancreatic stimulation, which is achieved by gastric suction. Special consideration for adequate fluid replacement is mandatory. The most common therapeutic error is underestimation of intravascular fluid loss. Management should also allow for recognition and correction of has received renewed support.