Article
Oxygen Therapy for Acute Myocardial Infarction Basis for a Practical Approach
Abstract
AbstractThe immediate reduction in myocardial strength after acute myocardial infarction may be the result of myocardial hypoxia and intracellular acidosis. Pulmonary venoarterial shunting, ventilation/perfusion imbalances, and diffusion abnormalities can reduce systemic arterial oxygen tension. Myocardial hypoxia and systemic hypoxia can perpetuate each other in a vicious cycle. Efforts that increase myocardial oxygen consumption can further the imbalance between myocardial oxygen supply and demand. Pure oxygen is suggested as therapy to increase myocardial oxygen tension after acute myocardial infarction, although there is some concern about oxygen toxicity. Despite lack of conclusive evidence that intramyocardial oxygenation is improved during oxygen therapy, there is evidence available to justify increasing arterial oxygen tension up to 100 mm Hg after acute myocardial infarction.This content is limited to qualifying members.
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